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etoposide eto  (MedChemExpress)


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    MedChemExpress etoposide eto
    Etoposide Eto, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 97/100, based on 216 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/etoposide+eto/Etoposide/pm41266351-277-43-45
    Average 97 stars, based on 216 article reviews
    etoposide eto - by Bioz Stars, 2026-10
    97/100 stars

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    other:

    Article Title: Engineered chromatin readers track damaged chromatin dynamics in live cells and animals
    Article Snippet: Where indicated, the following compounds were used at the indicated final concentrations, unless stated otherwise: 4-hydroxytamoxifen − 4-OHT (300 nM, Sigma–Aldrich – H6278), ATMi KU-55933 (10 μM, Selleckchem - S1092), Camptothecin-CPT (25 nM, Enzo Life Sciences - ALX-350-015), Cisplatin-CISP (10 μg/mL, Millipore − 232120), Etoposide-ETO (20 μM, MedChemExpress - HY-13629), Methylmethanesulfonate-MMS (0.25 mM, Sigma–Aldrich − 129925), Olaparib-OLAP (1 μM, Sigma–Aldrich - SML3705).

    Article Title: Engineered chromatin readers track damaged chromatin dynamics in live cells and animals.
    Article Snippet: Where indicated, the following compounds were used at the indicated final concentrations, unless stated otherwise: 4-hydroxytamoxifen − 4-OHT (300 nM, Sigma–Aldrich – H6278), ATMi KU-55933 (10 μM, Selleckchem - S1092), Camptothecin-CPT (25 nM, Enzo Life Sciences - ALX-350-015), Cisplatin-CISP (10μg/mL, Millipore − 232120), Etoposide-ETO (20μM, MedChemExpress - HY-13629), Methylmethanesulfonate-MMS (0.25mM, Sigma–Aldrich − 129925), Olaparib-OLAP (1μM, Sigma–Aldrich - SML3705).

    Article Title: MEN1 is a regulator of alternative splicing and prevents R-loop-induced genome instability through suppression of RNA polymerase II elongation.
    Article Snippet: After 24 h of incubaion, the cells wer e tr eated with various doses of Cislatin (15663–271, Solarbio), etoposide (ETO) (IE0270, Soarbio), N -methyl- N ’-nitr o- N -nitr osoguanidine (MNNG) 924–16-3, Sigma), mitomycin C (MMC) (GC12353, GLPIO), Madrasin (Mad) (HY-100236, MCE) or isoginkgetin Iso) (HY-N2117, MCE).

    Article Title: Probing DNA damage sites reveals context-dependent and novel DNA damage response factors
    Article Snippet: Where indicated, the following compounds were used at the indicated final concentrations, unless stated otherwise: ATMi KU-55933 (10 μM, Selleckchem, S1092), Camptothecin-CPT (25nM, Enzo Life Sciences - ALX-350-015), Cisplatin-CISP (10μg/mL, Millipore - 232120), Etoposide-ETO (20μM, MedChemExpress - HY-13629), Methyl methanesulfonate-MMS (0.25mM, Sigma-Aldrich - 129925), Olaparib-OLAP (1μM, Sigma-Aldrich - SML3705).



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    Pharmacological inhibition of ATM activation counteracts the elevated <t>ETO-induced</t> effects in brcc3 mutant embryos. ( A ) Schematic diagram of WT and brcc3 mutant embryos treated with ETO or ETO plus an ATM inhibitor. ( B ) Addition of an ATM <t>inhibitor</t> <t>KU60019</t> rescued the phenotypic abnormalities caused by ETO treatment. The proportion of embryos with the indicated phenotypes is shown in the bottom right corner of each panel. Scale bar = 500 μm. ( C ) Quantitative results of embryos with morphological normal as shown in ( B ). The total number of embryos is shown below the column. Values are represented as means ± s.d. *** p < 0.001; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test). ( D ) Acridine orange staining of WT and brcc3 mutant embryos at 28 hpf after ETO or ETO plus KU60019 treatment. White boxes indicate local magnification. Scale bar = 500 μm. ( E ) Quantitative results from images of head or 16–21 somite regions as shown in ( D ). The total number of embryos is shown above the column. Results are from three independent replicates. Values are represented as means ± s.d. ns, not significant; * p < 0.05; ** p < 0.01; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test).
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    Pharmacological inhibition of ATM activation counteracts the elevated <t>ETO-induced</t> effects in brcc3 mutant embryos. ( A ) Schematic diagram of WT and brcc3 mutant embryos treated with ETO or ETO plus an ATM inhibitor. ( B ) Addition of an ATM <t>inhibitor</t> <t>KU60019</t> rescued the phenotypic abnormalities caused by ETO treatment. The proportion of embryos with the indicated phenotypes is shown in the bottom right corner of each panel. Scale bar = 500 μm. ( C ) Quantitative results of embryos with morphological normal as shown in ( B ). The total number of embryos is shown below the column. Values are represented as means ± s.d. *** p < 0.001; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test). ( D ) Acridine orange staining of WT and brcc3 mutant embryos at 28 hpf after ETO or ETO plus KU60019 treatment. White boxes indicate local magnification. Scale bar = 500 μm. ( E ) Quantitative results from images of head or 16–21 somite regions as shown in ( D ). The total number of embryos is shown above the column. Results are from three independent replicates. Values are represented as means ± s.d. ns, not significant; * p < 0.05; ** p < 0.01; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test).
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    Image Search Results


    Pharmacological inhibition of ATM activation counteracts the elevated ETO-induced effects in brcc3 mutant embryos. ( A ) Schematic diagram of WT and brcc3 mutant embryos treated with ETO or ETO plus an ATM inhibitor. ( B ) Addition of an ATM inhibitor KU60019 rescued the phenotypic abnormalities caused by ETO treatment. The proportion of embryos with the indicated phenotypes is shown in the bottom right corner of each panel. Scale bar = 500 μm. ( C ) Quantitative results of embryos with morphological normal as shown in ( B ). The total number of embryos is shown below the column. Values are represented as means ± s.d. *** p < 0.001; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test). ( D ) Acridine orange staining of WT and brcc3 mutant embryos at 28 hpf after ETO or ETO plus KU60019 treatment. White boxes indicate local magnification. Scale bar = 500 μm. ( E ) Quantitative results from images of head or 16–21 somite regions as shown in ( D ). The total number of embryos is shown above the column. Results are from three independent replicates. Values are represented as means ± s.d. ns, not significant; * p < 0.05; ** p < 0.01; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test).

    Journal: International Journal of Molecular Sciences

    Article Title: Loss of Brcc3 in Zebrafish Embryos Increases Their Susceptibility to DNA Damage Stress

    doi: 10.3390/ijms252212108

    Figure Lengend Snippet: Pharmacological inhibition of ATM activation counteracts the elevated ETO-induced effects in brcc3 mutant embryos. ( A ) Schematic diagram of WT and brcc3 mutant embryos treated with ETO or ETO plus an ATM inhibitor. ( B ) Addition of an ATM inhibitor KU60019 rescued the phenotypic abnormalities caused by ETO treatment. The proportion of embryos with the indicated phenotypes is shown in the bottom right corner of each panel. Scale bar = 500 μm. ( C ) Quantitative results of embryos with morphological normal as shown in ( B ). The total number of embryos is shown below the column. Values are represented as means ± s.d. *** p < 0.001; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test). ( D ) Acridine orange staining of WT and brcc3 mutant embryos at 28 hpf after ETO or ETO plus KU60019 treatment. White boxes indicate local magnification. Scale bar = 500 μm. ( E ) Quantitative results from images of head or 16–21 somite regions as shown in ( D ). The total number of embryos is shown above the column. Results are from three independent replicates. Values are represented as means ± s.d. ns, not significant; * p < 0.05; ** p < 0.01; **** p < 0.0001 (one-way ANOVA followed by Tukey’s post hoc test).

    Article Snippet: ETO (HY-13629), KU60019 (HY-12061), and CGK733 (HY-15520) were purchased from MedChemExpress (Monmouth, NJ, USA).

    Techniques: Inhibition, Activation Assay, Mutagenesis, Staining